| Begin | 915 | Description | In pen3-2; overaccumulation in leaves of 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) upon Blumeria graminis conidiospore inoculation, a pathogen-inducible tryptophan-derived compound, which biosynthesis is dependent on the PEN2 metabolic pathway. More susceptible to the necrotrophic pathogen P. cucumerina, with higher frequency of fungal penetration and increased formation of elongating secondary hyphae after the first haustorium development. Fully defective in preinvasive defense to nonadapted powdery mildews (e.g. Blumeria graminis and Erysiphe pisi). Extensive leaf chlorosis and reduced fungal growth upon infection by the host-adapted pathogen Golovinomyces orontii associated with an hyperaccumulation of both free and total salicylic acid (SA) as well as pathogen-inducible hydrogen peroxides in leaves. Impaired microbe-associated molecular patterns (MAMPs)-induced (e.g. flg22) callose deposition. Hypersensitivity to root growth inhibition by indole 3-butyric acid (IBA), an auxin precursor. Reduced accumulation (at protein level). |
| End | 915 | Type | mutagenesis site |
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