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Search results 1 to 32 out of 32 for Microbe-associated molecular pattern

Category restricted to UniProtFeature (x)

0.033s

Categories

Category: UniProtFeature
Type Details Score
UniProt Feature
Begin: 317
Description: In fkd2-2; vein pattern defects.
Type: mutagenesis site
End: 317
UniProt Feature
Begin: 518
Description: In scf-6; intermediate vein pattern defects.
Type: mutagenesis site
End: 518
UniProt Feature
Begin: 519
Description: In scf-1; strong vein pattern defects.
Type: mutagenesis site
End: 519
UniProt Feature
Begin: 1
Description: Pathogen-associated molecular patterns-induced protein A70
Type: chain
End: 379
UniProt Feature
Begin: 196
Description: No effect on molecular weight.
Type: mutagenesis site
End: 196
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 7
Type: chain
End: 446
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 2
Type: chain
End: 285
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 1
Type: chain
End: 342
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 3
Type: chain
End: 303
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 6
Type: chain
End: 1043
UniProt Feature
Begin: 1
Description: BAG family molecular chaperone regulator 4
Type: chain
End: 269
UniProt Feature
Begin: 15
Description: BAG family molecular chaperone regulator 5, mitochondrial
Type: chain
End: 215
UniProt Feature
Begin: 53
Description: BAG family molecular chaperone regulator 8, chloroplastic
Type: chain
End: 551
UniProt Feature
Begin: 189
Description: Phospho-mimetic sequence variation. Normal transcriptional repressor activity but enhanced DNA-binding affinity leading to compromised microbe-associated molecular patterns- (MAMPs) triggered immunity (PTI) responses.
Type: mutagenesis site
End: 189
UniProt Feature
Begin: 407
Description: In gct-3; uncoupled cell division, pattern formation and morphogenesis during embryogenesis.
Type: mutagenesis site
End: 407
UniProt Feature
Begin: 321
Description: In van3-2 and fkd2-1; reduced ARFGAP activity, mis-localization and vein pattern defects.
Type: mutagenesis site
End: 321
UniProt Feature
Begin: 15
Description: No effect on molecular weight; when associated with V-109 and V-116.
Type: mutagenesis site
End: 15
UniProt Feature
Begin: 116
Description: No effect on molecular weight; when associated with V-15 and V-109.
Type: mutagenesis site
End: 116
UniProt Feature
Begin: 109
Description: No effect on molecular weight; when associated with V-15 and V-116.
Type: mutagenesis site
End: 109
UniProt Feature
Begin: 138
Description: Decreases the molecular weight of OSCA1 when overexpressed in a heterologous system.
Type: mutagenesis site
End: 138
UniProt Feature
Begin: 124
Description: No effect on the molecular weight of OSCA1 when overexpressed in a heterologous system.
Type: mutagenesis site
End: 124
UniProt Feature
Begin: 1357
Description: Required for preinvasive defense to nonadapted powdery mildews but not for salicylic acid (SA)- and microbe-associated molecular patterns (MAMPs)- dependent defense against host-adapted pathogens. Necessary to prevent the sensitivity to IBA (an auxin precursor)
Type: site
End: 1357
UniProt Feature
Begin: 354
Description: Required for both preinvasive defense to nonadapted powdery mildews and salicylic acid (SA)-, microbe-associated molecular patterns (MAMPs)- dependent defense against host-adapted pathogens, and defense responses toward necrotrophic pathogens. Necessary to prevent the sensitivity to IBA (an auxin precursor)
Type: site
End: 354
UniProt Feature
Begin: 915
Description: Required for both preinvasive defense to nonadapted powdery mildews and salicylic acid (SA)-, microbe-associated molecular patterns (MAMPs)- dependent defense against host-adapted pathogens, and defense responses toward necrotrophic pathogens. Necessary to prevent the sensitivity to IBA (an auxin precursor)
Type: site
End: 915
UniProt Feature
Begin: 704
Description: Required for preinvasive defense to nonadapted powdery mildews but not for salicylic acid (SA)- and microbe-associated molecular patterns (MAMPs)- dependent defense against host-adapted pathogens. Involved in defense responses toward necrotrophic pathogens. Not necessary to prevent the sensitivity to IBA (an auxin precursor)
Type: site
End: 704
UniProt Feature
Begin: 70
Description: In cce5-1; reduced calcium levels after elicitation with peptides representing bacteria-derived microbe- and damage-associated molecular patterns (MAMPs, flg22 and elf18, and the endogenous DAMPAtPep1), but normal response to chitin octamers. Impaired phosphorylation upon flagellin (flg22) treatment, and lost kinase activity and autophosphorylation.
Type: mutagenesis site
End: 70
UniProt Feature
Begin: 97
Description: In cce5-3; reduced calcium levels after elicitation with peptides representing bacteria-derived microbe- and damage-associated molecular patterns (MAMPs, flg22 and elf18, and the endogenous DAMPAtPep1), but normal response to chitin octamers. Impaired phosphorylation upon flagellin (flg22) treatment, and lost kinase activity and autophosphorylation.
Type: mutagenesis site
End: 97
UniProt Feature
Begin: 172
Description: In cce5-5; reduced calcium levels after elicitation with peptides representing bacteria-derived microbe- and damage-associated molecular patterns (MAMPs, flg22 and elf18, and the endogenous DAMPAtPep1), but normal response to chitin octamers. Impaired phosphorylation upon flagellin (flg22) treatment, and lost kinase activity and autophosphorylation.
Type: mutagenesis site
End: 172
UniProt Feature
Begin: 704
Description: In pen3-5; slight overaccumulation in leaves of 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) upon Blumeria graminis conidiospore inoculation, a pathogen-inducible tryptophan-derived compound, which biosynthesis is dependent on the PEN2 metabolic pathway. Fully defective in preinvasive defense to nonadapted powdery mildews (e.g. Blumeria graminis and Erysiphe pisi). Normal susceptibility to the host-adapted pathogen Golovinomyces orontii associated with normal salicylic acid (SA) pathogen-inducible hydrogen peroxides levels. Retained microbe-associated molecular patterns (MAMPs)-induced (e.g. flg22) callose deposition. Increased susceptibility to the necrotrophic pathogen Plectosphaerella cucumerina. Enhancer mutation of the mutant pen2-dependent invasive growth of Blumeria graminis. Normal insensitivity to indole-3-butyric acid (IBA), an auxin precursor. Normal accumulation (at protein level).
Type: mutagenesis site
End: 704
UniProt Feature
Begin: 1357
Description: In pdr8-115 and pen3-6; slight overaccumulation in leaves of 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) upon Blumeria graminis conidiospore inoculation, a pathogen-inducible tryptophan-derived compound, which biosynthesis is dependent on the PEN2 metabolic pathway. Hypersensitivity to root growth inhibition by 2,4-dichlorophenoxybutyric acid (2,4-DB), an analog of indole 3-butyric acid (IBA), an auxin precursor. Fully defective in preinvasive defense to nonadapted powdery mildews (e.g. Blumeria graminis and Erysiphe pisi). Normal susceptibility to the host-adapted pathogen Golovinomyces orontii associated with normal salicylic acid (SA), and pathogen-inducible hydrogen peroxides levels. Retained microbe-associated molecular patterns (MAMPs)-induced (e.g. flg22) callose deposition. Suppressor of a subset of ibr5 mutant phenotypes including IBA sensitivity but without suppressing ibr5 resistance to indole-3-acetic acid (IAA). Normal accumulation (at protein level).
Type: mutagenesis site
End: 1357
UniProt Feature
Begin: 354
Description: In pen3-1; overaccumulation in leaves of 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) upon Blumeria graminis conidiospore inoculation, a pathogen-inducible tryptophan-derived compound, which biosynthesis is dependent on the PEN2 metabolic pathway. More susceptible to the necrotrophic pathogen Plectosphaerella cucumerina, with higher frequency of fungal penetration and increased formation of elongating secondary hyphae after the first haustorium development. Fully defective in preinvasive defense to nonadapted powdery mildews (e.g. Blumeria graminis and Erysiphe pisi). Extensive leaf chlorosis and reduced fungal growth upon infection by the host-adapted pathogen Golovinomyces orontii associated with an hyperaccumulation of both free and total salicylic acid (SA) as well as pathogen-inducible hydrogen peroxides in leaves. Impaired microbe-associated molecular patterns (MAMPs)-induced (e.g. flg22) callose deposition. Hypersensitivity to root growth inhibition by indole 3-butyric acid (IBA), an auxin precursor. Reduced accumulation (at protein level).
Type: mutagenesis site
End: 354
UniProt Feature
Begin: 915
Description: In pen3-2; overaccumulation in leaves of 4-O-beta-D-glucosyl-indol-3-yl formamide (4OGlcI3F) upon Blumeria graminis conidiospore inoculation, a pathogen-inducible tryptophan-derived compound, which biosynthesis is dependent on the PEN2 metabolic pathway. More susceptible to the necrotrophic pathogen P. cucumerina, with higher frequency of fungal penetration and increased formation of elongating secondary hyphae after the first haustorium development. Fully defective in preinvasive defense to nonadapted powdery mildews (e.g. Blumeria graminis and Erysiphe pisi). Extensive leaf chlorosis and reduced fungal growth upon infection by the host-adapted pathogen Golovinomyces orontii associated with an hyperaccumulation of both free and total salicylic acid (SA) as well as pathogen-inducible hydrogen peroxides in leaves. Impaired microbe-associated molecular patterns (MAMPs)-induced (e.g. flg22) callose deposition. Hypersensitivity to root growth inhibition by indole 3-butyric acid (IBA), an auxin precursor. Reduced accumulation (at protein level).
Type: mutagenesis site
End: 915